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We were unable to process your request. Please try again later. If you continue to have this issue please contact customerservice@slackinc.com.<\/strong> <\/p>\nBack to Healio<\/button><\/p>\n<\/div>\n<\/div><\/div>\n<\/p><\/div>\n<\/p><\/div>\n<\/div>\nKey takeaways:<\/h2>\n\nParticipants were aged 70 years and older with moderate hearing impairment and no dementia. <\/li>\n Estimated mean cognition scores for participants with and without hearing aid prescriptions at 7 years were similar. <\/li>\n<\/ul>\nHearing aids did not lead to any significant differences in cognitive testing among older patients with moderate hearing loss, but these devices may reduce risks for dementia, according to data published in Neurology<\/i>. <\/p>\n
Residual confounding may explain these findings, Lachlan Cribb,<\/b> BSci,<\/b> MEpi,<\/b> a PhD student at the School of Public Health and Preventive Medicine, Monash University, Melbourne, Australia, and colleagues wrote. <\/p>\n
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Neurology<\/i>. 2026;doi:10.1212\/WNL.0000000000214572.
\u201cHearing loss has been linked to a greater risk of developing dementia. But it was not known whether using a hearing aid could help reduce that risk,\u201d Cribb told Healio. <\/p>\n
The 2023 randomized Aging and Cognitive Health Evaluation in Elders (ACHIEVE) trial compared a hearing aid intervention with health education among older adults in the United States with hearing loss. <\/p>\n
\u201cThey found little overall benefit of hearing aids for slowing cognitive decline over 3 years, though there was a benefit in a subgroup with elevated baseline risk,\u201d Cribb said. <\/p>\n
\u201cDue to its relatively short follow-up, diagnosed dementia was not examined,\u201d he continued. \u201cThe goal of our study was to investigate the effects of hearing aids on dementia and cognition over a period of 7 years.\u201d<\/p>\n
The study examined data from patients aged 70 years and older with moderate hearing impairment but no dementia. All patients were enrolled in the ASPirin in Reducing Events in the Elderly (ASPREE) randomized, placebo-controlled trial and its ASPREE-eXTension follow-up. <\/p>\n
The cohort included a median of 664 patients (mean age, 75.1 years; 49% women; 98% white) who received a prescription for a new hearing aid in the previous 3 years, including 402 who always or often used their hearing aid and 188 who sometimes or rarely used it, and a median of 2,113 (mean age, 74.2 years; 48% women; 99% white) who did not. <\/p>\n
At 7 years, the subgroup with prescriptions included 51 deaths, 25 cases of dementia and 131 cases of cognitive impairment, and the subgroup with no prescriptions included 123 deaths, 92 cases of dementia and 411 cases of cognitive impairment. <\/p>\n
\u201cAfter carefully adjusting for a range of lifestyle and health factors that differed across participants, we found that participants with hearing loss who were prescribed hearing aids had a similar rate of cognitive change over 7 years to those who were not prescribed hearing aids,\u201d Cribb said.<\/p>\n
Estimated mean scores across four cognitive tests at 7 years for these subgroups had a mean difference of 0.03 standard deviation [SD] (95% CI, \u20130.14-0.21), the researchers said, and the estimated mean scores for those who often or always used hearing aids were similar to the scores for those who never used hearing aids as well (mean difference = 0.02 SD; 95% CI, \u20130.19-0.23). <\/p>\n
Estimated risks for dementia at 7 years included 5% for those with a prescription for a hearing aid and 7.5% for those with no prescription (RR = 0.67; 95% CI, 0.37-0.97).<\/p>\n
\u201cThose who were prescribed hearing aids had a 33% lower risk of diagnosed dementia, with the degree of risk reduction proportional to how frequently participants reported using their hearing aid,\u201d Cribb said. <\/p>\n
Specifically, these risks included 7.3% for those who never used a hearing aid, 6.8% with rare or sometimes use and 4.8% for those who used a hearing aid often or always. <\/p>\n
The risk ratio for dementia was 0.67 (95% CI, 0.29-1.06) among those who used a hearing aid often or always compared with those who never used one. <\/p>\n
Estimated risks for cognitive impairment at 7 years included 36.1% for those with a prescription for a hearing aid and 42.4% for those with no prescription (RR = 0.85; 95% CI, 0.7-1), or a 15% lower risk with a prescription. <\/p>\n
Specific risks included 41.5% for those who never used a hearing aid, 39.2% for those with rare or sometimes use and 35.7% for those who often or always used a hearing aid. <\/p>\n
The risk ratio for cognitive impairment among patients who often or always used a hearing aid was 0.86 (95% CI, 0.66-1.06) compared with those who never used one. <\/p>\n
Further, mean differences in overall cognition scores at 7 years were greater for patients with an overall baseline Modified Min-Mental State Examination score less than 95 (0.17 SD; 95% CI, \u20130.07-0.4) and baseline better-ear four-frequency pure tone average of 30 dbHL or higher (0.12 SD; 95% CI, \u20130.12-0.36).<\/p>\n
As a sensitivity analysis, the researchers compared the estimated 3-year mean differences in overall cognition scores between patients who often or always used hearing aids and those who never used them with the effect estimate from the ACHIEVE trial. These were 0.04 standard deviations (95% CI, \u20130.05-0.13) and 0 standard deviations (95% CI, \u20130.08-0.08), respectively, which the researchers called similar. <\/p>\n
Further, the estimated 7-year mean difference in overall cognition scores between those patients with a prescription and those with no prescription was greater (0.14 SD; 95% CI, \u20130.08-0.35) when multiply imputed audiometry data were used to define the eligibility criterion of moderate hearing loss. Overall risks for dementia and cognitive impairment were considerably greater when audiometry was used to define the eligible sample. <\/p>\n
Based on these findings, the researchers said that prescriptions for hearing aids did not lead to any substantial changes in overall cognition scores for older patients with moderate hearing impairment. <\/p>\n
\u201cWe were surprised to find evidence of reduced dementia risk without a corresponding reduction in overall age-related cognitive change,\u201d Cribb said. <\/p>\n
\u201cWe postulate that this could be due to the good cognitive health of our sample at baseline (reducing scope for treatment benefit) and\/or to the complexity of age-related cognitive change, which is influenced by a diverse range of lifestyle and health factors,\u201d he continued.<\/p>\n
The researchers said these findings may be due to possible links between sensory loss and dementia risk. As neuronal activity altered by hearing loss may exacerbate existing pathology in Alzheimer\u2019s disease, they said, amplification may delay or prevent dementia onset for these patients but have less benefit for patients who do not have this pathology. <\/p>\n
They added that the findings may support the hypothesis that hearing loss causes sensory deprivation, impacting regional brain volume. <\/p>\n
Cribb and colleagues called for long-term randomized trials and observational studies in populations who are at a high risk for cognitive decline. Overall, Cribb said, the use of hearing aids to treat hearing loss could reduce risks for dementia in a dose-dependent manner. <\/p>\n
\u201cWe found that treating hearing loss with hearing aids could reduce dementia risk in a dose-dependent manner,\u201d he said.<\/p>\n
\u201cWhile we can\u2019t conclude that hearing aids cause a reduction in dementia, together with the findings of ACHIEVE, this suggests that screening and treatment for hearing loss may be a low-risk and cost-effective way of reducing dementia risk,\u201d he added. <\/p>\n
\u201cThis may be especially true for individuals with risk factors for cognitive impairment, including preexisting cognitive difficulties, limited education, and cardiovascular and metabolic comorbidities.\u201d <\/p>\n
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These findings are significant, particularly due to the robust \u201ctarget trial\u201d emulation design. They also highlight a notable divergence. While hearing aids did not substantially change cognitive scores, they were associated with an approximately 33% reduction in dementia risk. This suggests that while hearing aids may not boost performance on cognitive tests, they may act as a crucial buffer against the threshold of clinical dementia.<\/default:p>While the results align with the consensus that treating hearing loss is protective, further studies should include a critical variable: central hearing. The authors relied on peripheral measures like self-reports and pure tone averages, which measure the ear\u2019s ability to detect sound, not the brain\u2019s ability to process it. <\/default:p>In my experience, central auditory dysfunction is common in early Alzheimer\u2019s. Simply amplifying sound with a hearing aid does not fix this neural deficit, which may explain why general cognitive scores did not improve despite the lower dementia risk.<\/default:p>Clinicians should feel confident recommending hearing aids earlier, as the study showed that \u201coften\/always\u201d using them was associated with lower dementia risk compared with \u201cnever\u201d using them. <\/default:p>However, clinicians must look beyond the audiogram. If a patient has hearing aids but still struggles to understand speech in noise, it likely indicates a central processing issue requiring cognitive rehabilitation strategies beyond simple amplification.<\/default:p>Future research should measure central auditory function. We need to disentangle whether hearing aids prevent dementia by restoring input, or if they are simply less effective in people whose \u201chearing loss\u201d is actually a symptom of early brain failure. Measuring this variable is essential to predict which patients will benefit most.<\/default:p><\/p>\n\n
Meher Lad, MBBS, PhD<\/strong><\/p>\n Newcastle upon Tyne Hospitals NHS Foundation Trust<\/span><\/div>\n
Disclosures: <\/strong> Lad reports no relevant financial disclosures.
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Overall, these findings add to evidence suggesting that hearing aids use may be associated with a reduced risk for dementia and cognitive impairment. However, what was surprising was the estimated rates of cognitive decline were similar between those with and without hearing aids. <\/default:p>There are not clear biologic mechanisms to support this finding since cognitive decline and impairment is a key component of dementia diagnosis. It is more likely that one analysis is due to methodologic differences or some sort of bias impacting one analysis more than the other.<\/default:p>This work is still interesting because it used innovative and rigorous methods that can help account for some types of bias and is more directly comparable to trial results.<\/default:p>Also, both these sets of findings match prior data, which has been mixed in this topic. There is growing evidence that hearing aids are associated with a reduced risk for dementia, but mechanisms are still unknown.<\/default:p>While this study and others have promising evidence that hearing aids may help reduce risk for dementia, I think this study points out that we may not fully understand the association between hearing loss, hearing aid use, and dementia. Although this study used innovative and robust methods to emulate a trial, this design cannot fully account for potential biasing factors. <\/default:p>I would caution clinicians from drawing too firm conclusions from these findings. However, we know hearing aids can benefit adults with hearing loss in other ways, so clinicians may discuss hearing with older patients and refer them to get their hearing checked if it is a potential concern.<\/default:p>Health care could benefit from more long-term studies on hearing aid use with repeat measures over time and more studies that could test whether the quality of hearing with hearing aids and use patterns are associated with dementia risk. More studies are needed to examine potential mechanisms of the relationship with dementia, such as the role of social connections and communications or associations with biomarkers for Alzheimer\u2019s disease. <\/default:p><\/p>\n\n
Willa Brenowitz, PhD, MPH<\/strong><\/p>\n Kaiser Permanente Northwest Center for Health Research<\/span><\/div>\n
Disclosures: <\/strong> Brenowitz reports no relevant financial disclosures.
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Sources\/Disclosures<\/h3>\n Source: <\/h2>\n
Neurology<\/i>. 2026;doi:10.1212\/WNL.0000000000214572. <\/p>\nReferences:<\/h2>\n
For more information:<\/b> <\/p>\n
Lachlan Cribb,<\/b> BSci,<\/b> MEpi<\/b>, <\/b>can be reached at allergy@healio.com.<\/p>\n\n
Disclosures: <\/strong>
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